WATTYL KILLRUST GLASSFLAKE POLYESTER PART B
Flammability | 0 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 2 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Part B of a two part glassflake polyester coating.
"methyl ethyl ketone peroxide in phthalate plasticiser", "curing agent for unsaturated
polyester resins", Polychem
Contact with combustible material may cause fire.
Irritating to eyes.
Harmful by inhalation and if swallowed.
Repeated exposure may cause skin dryness and cracking.
Very toxic to aquatic organisms, may cause long- term adverse effects in the
aquatic environment.
Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. Considered an unlikely route of entry in commercial/industrial environments. Ingestion may result in nausea, abdominal irritation, pain and vomiting. Toxic myocarditis may follow ingestion of oxidizing agents such as peroxides. -------------------------------------------------------------- BASIC TREATMENT -------------------------------------------------------------- · Establish a patent airway with suction where necessary. · Watch for signs of respiratory insufficiency and assist ventilation as necessary. · Administer oxygen by non-rebreather mask at 10 to 15 l/min. · Monitor and treat, where necessary, for pulmonary edema . · Monitor and treat, where necessary, for shock. · Anticipate seizures . · DO NOT use emetics. Where ingestion is suspected rinse mouth and give up to 200 ml water (5 ml/kg recommended) for dilution where patient is able to swallow, has a strong gag reflex and does not drool. · DO NOT attempt neutralization as exothermic reaction may occur. · Skin burns should be covered with dry, sterile bandages, following decontamination. -------------------------------------------------------------- ADVANCED TREATMENT -------------------------------------------------------------- · Consider orotracheal or nasotracheal intubation for airway control in unconscious patient or where respiratory arrest has occurred. · Positive-pressure ventilation using a bag-valve mask might be of use. · Monitor and treat, where necessary, for arrhythmias. · Start an IV D5W TKO. If signs of hypovolemia are present use lactated Ringers solution. Fluid overload might create complications. · Drug therapy should be considered for pulmonary edema. · Hypotension with signs of hypovolemia requires the cautious administration of fluids. Fluid overload might create complications. · Treat seizures with diazepam. · Proparacaine hydrochloride should be used to assist eye irrigation. BRONSTEIN, A.C. and CURRANCE, P.L. EMERGENCY CARE FOR HAZARDOUS MATERIALS EXPOSURE: 2nd Ed. 1994.
This material can cause eye irritation and damage in some persons.
The material is not thought to produce adverse health effects or skin irritation following contact (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable gloves be used in an occupational setting. Bare unprotected skin should not be exposed to this material.
There is some evidence to suggest that this material, if inhaled, can irritate the throat and lungs of some persons. Not normally a hazard due to non-volatile nature of product. Inhalation hazard is increased at higher temperatures. Inhalation of quantities of liquid mist may be extremely hazardous, even lethal due to spasm, extreme irritation of larynx and bronchi, chemical pneumonitis and pulmonary edema. Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.
Principal routes of exposure are usually by skin contact with the liquid and eye contact with the liquid and inhalation of vapor/spray mist. The material may accumulate in the human body and progressively causetissue damage. Chronic exposure by rats repeatedly dosed with MEKP 3 times/weekly for 7 weeks by the intraperitoneal or oral route (13 mg/kg and 97 mg/kg respectively) produced marked evidence of cumulative toxicity. The liver showed occasional damage, consisting of fatty degeneration in the central portion of the lobule and an increased number of round cells in the portal spaces; the proximal tubules of the kidney showed desquamation of the epithelium whilst the convoluted tubules showed granular precipitates or castes in the lumina.