LAURYLDIMETHYLETHYLAMMONIUM ETHYLSULFATE
Flammability | 1 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 0 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Antistatic. Quaternary ammonium compound.
C16-H36-N.C2-H5-O4-S, "1-dodecanaminium, N-ethyl-N, N-dimethyl-, ethyl sulfate", "1-
dodecanaminium, N-ethyl-N, N-dimethyl-, ethyl sulfate", "lauryldimethylethylammonium
ethosulfate", "dodecyldimethylethylammonium, ethosulfate", "lauryldimethylethylammonium
ethyl sulfate", "N-ethyl-N, N, -dimethyl-1-dodecaminium ethyl sulfate", "N-ethyl-N, N, -
dimethyl-1-dodecaminium ethyl sulfate"
Harmful if swallowed.
Toxic to aquatic organisms.
Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. Concentrated solutions of many cationics may cause corrosive damage to mucous membranes and the esophagus. Nausea and vomiting (sometimes bloody) may follow ingestion. Serious exposures may produce an immediate burning sensation of the mouth, throat and abdomen with profuse salivation, ulceration of mucous membranes, signs of circulatory shock (hypotension, labored breathing, and cyanosis) and a feeling of apprehension, restlessness, confusion and weakness. Weak convulsive movements may precede central nervous system depression. Erosion, ulceration, and petechial hemorrhage may occur through the small intestine with glottic, brain and pulmonary edema. Death may result from asphyxiation due to paralysis of the muscles of respiration or cardiovascular collapse. Fatal poisoning may arise even when the only pathological signs are visceral congestion, swallowing, mild pulmonary edema or varying signs of gastrointestinal irritation. Individuals who survive a period of severe hypertension may develop kidney failure. Cloudy swelling, patchy necrosis and fatty infiltration in such visceral organs as the heart, liver and kidneys shows at death.
Although the material is not thought to be an irritant, direct contact with the eye may produce transient discomfort characterized by tearing or conjunctival redness (as with windburn). Concentrated solutions of cationic surfactants may cause destruction of the tissue lining the mouth, throat and gullet, and may cause nausea and vomiting. In sufficient quantity may produce restlessness, confusion, low blood pressure, muscle weakness, collapse, convulsion, labored breathing, blue coloration of the lips and coma. Death may occur in 1- 4 hours. Fatal dose is estimated at 1-3 grams for certain cationics.
The material is not thought to produce adverse health effects or skin irritation following contact (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable gloves be used in an occupational setting. Toxic effects may result from skin absorption. Many cationic surfactants are very irritating to the eyes at low concentration. Concentrated solutions can cause severe burns with permanent clouding.
The material is not thought to produce adverse health effects or irritation of the respiratory tract (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable control measures be used in an occupational setting. Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled. Respiratory sensitization may result in allergic/asthma like responses; from coughing and minor breathing difficulties to bronchitis with wheezing, gasping. Exposure to Sulfonates can cause an imbalance in cellular salts and therefore cellular function. Airborne sulfonates may be responsible for respiratory allergies and, in some instances, minor dermal allergies.
There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population.
Principal routes of exposure are by accidental skin and eye contact andinhalation of generated dusts. Alkyl-substituted sulfonates are thought to induce genetic mutations in cells.