LAURYLETHYLMORPHOLINIUM ETHOSULFATE
Flammability | 1 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 0 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Surfactant. Quaternary ammonium compound.
C20-H43-N-O5-S, C20-H43-N-O5-S, "morpholinium, 4-ethyl-4-dodecyl-, ethyl sulfate",
"morpholinium, 4-ethyl-4-dodecyl-, ethyl sulfate", "4-ethyl-4-dodecylmorpholinium ethyl
sulfate", "4-ethyl-4-dodecylmorpholinium ethyl sulfate", "N-lauryl-N-ethylmorpholinium
ethosulfate", "N-lauryl-N-ethylmorpholinium ethosulfate", "N-dodecyl-N-ethylmorpholinium
ethosulfate", "N-dodecyl-N-ethylmorpholinium ethosulfate", "quaternary ammonium compounds"
Harmful if swallowed.
Irritating to eyes and skin.
Toxic to aquatic organisms.
Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. Concentrated solutions of many cationics may cause corrosive damage to mucous membranes and the esophagus. Nausea and vomiting (sometimes bloody) may follow ingestion. Serious exposures may produce an immediate burning sensation of the mouth, throat and abdomen with profuse salivation, ulceration of mucous membranes, signs of circulatory shock (hypotension, labored breathing, and cyanosis) and a feeling of apprehension, restlessness, confusion and weakness. Weak convulsive movements may precede central nervous system depression. Erosion, ulceration, and petechial hemorrhage may occur through the small intestine with glottic, brain and pulmonary edema. Death may result from asphyxiation due to paralysis of the muscles of respiration or cardiovascular collapse. Fatal poisoning may arise even when the only pathological signs are visceral congestion, swallowing, mild pulmonary edema or varying signs of gastrointestinal irritation. Individuals who survive a period of severe hypertension may develop kidney failure. Cloudy swelling, patchy necrosis and fatty infiltration in such visceral organs as the heart, liver and kidneys shows at death. Sulfates are not well absorbed orally, but can cause diarrhea.
This material can cause eye irritation and damage in some persons. The material may produce severe irritation to the eye causing pronounced inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.
This material can cause inflammation of the skin oncontact in some persons. Skin contact is not thought to have harmful health effects, however the material may still produce health damage following entry through wounds, lesions or abrasions. Toxic effects may result from skin absorption. The material may cause severe skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin. Repeated exposures may produce severe ulceration.
The material is not thought to produce adverse health effects or irritation of the respiratory tract (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable control measures be used in an occupational setting. Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled. Respiratory sensitization may result in allergic/asthma like responses; from coughing and minor breathing difficulties to bronchitis with wheezing, gasping. Exposure to Sulfonates can cause an imbalance in cellular salts and therefore cellular function. Airborne sulfonates may be responsible for respiratory allergies and, in some instances, minor dermal allergies.
Principal routes of exposure are by accidental skin and eye contact andinhalation of generated dusts. Alkyl-substituted sulfonates are thought to induce genetic mutations in cells.