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WIA HOBART H800 MSDS报告[下载][中文版]

Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

WIA HOBART H800

NFPA

Flammability 0
Toxicity 2
Body Contact 2
Reactivity 0
Chronic 3
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4

PRODUCT USE

Flux powder.

SYNONYMS

"arc flux", "H 800"

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Irritating to eyes and skin.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be damaging to the health of the individual.  Magnesium salts are generally absorbed so slowly that oral administration causes few toxic effects, as the dose is readily expelled via the bowel. If evacuation fails, mucosal irritation and absorption may result. This can result in nervous system depression, heart effects, loss of reflexes and death due to paralysis of breathing. These usually do not occur unless the bowel or kidneys are damaged.  Poisonings rarely occur after oral administration of manganese salts because they are poorly absorbed from the gut.  Fluoride causes severe loss of calcium in the blood, with symptoms appearing several hours later including painful and rigid muscle contractions of the limbs. Cardiovascular collapse can occur and may cause death with increased heart rate and other heart rhythm irregularities. The brain and kidneys may be affected. Other toxic effects include headache, increased saliva output, jerking of the eyeball and dilated pupils, lethargy, stupor, coma and rarely, convulsions.  

EYE

  This material can cause eye irritation and damage in some persons.  Contact with the eye, by metal dusts, may produce mechanical abrasion or foreign body penetration of the eyeball. Iron particles embedded in the eye may produce a condition known as ocular siderosis; effects include discolouration of the cornea and iris and pupillary effects such as poor reaction to light and accommodation. Particles entering the lens may produce cataracts. A rare consequence of ocular siderosis is glaucoma.  

SKIN

  This material can cause inflammation of the skin oncontact in some persons.  The material may accentuate any pre-existing dermatitis condition.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  

INHALED

  Inhalation may produce health damage*.  Inhalation of dusts, generated by the material during the course of normal handling, may be damaging to the health of the individual.  Acute silicosis occurs under conditions of extremely high silica dust exposure particularly when the particle size of the dust is small. The disease is rapidly progressive and spreads widely through the lungs within months of the initial exposure and causing deaths within 1 to 2 years.  Effects on lungs are significantly enhanced in the presence of respirableparticles.  

CHRONIC HEALTH EFFECTS

  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  This material can cause serious damage if one is exposed to it for long periods. It can be assumed that it contains a substance which can produce severe defects. This has been demonstrated via both short- and long-term experimentation.  On the basis of epidemiological data, it has been concluded that prolonged inhalation of the material, in an occupational setting, may produce cancer in humans.  Exposure to large doses of Aluminum has been connected with the degenerative brain disease Alzheimer's Disease.  Manganese is an essential trace element. Chronic exposure to low levels of manganese can include a mask-like facial expression, spastic gait, tremors, slurred speech, disordered muscle tone, fatigue, anorexia, loss of strength and energy, apathy and poor concentration.  Repeated exposures, in an occupational setting, to high levels of fine- divided dusts may produce a condition known as pneumoconiosis which is the lodgement of any inhaled dusts in the lung irrespective of the effect. This is particularly true when a significant number of particles less than 0.5 microns (1/50,000 inch), are present. Lung shadows are seen in the X-ray. Symptoms of pneumoconiosis may include a progressive dry cough, shortness of breath on exertion, increased chest expansion, weakness and weight loss. As the disease progresses the cough produces a stringy mucous, vital capacity decreases further and shortness of breath becomes more severe. Pneumoconiosis is the accumulation of dusts in the lungs and the tissue reaction in its presence. It is further classified as being of noncollagenous or collagenous types. Noncollagenous pneumoconiosis, the benign form, is identified by minimal stromal reaction, consists mainly of reticulin fibres, an intact alveolar architecture and is potentially reversible.  
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