JASOL PROTREAT 363
Flammability | 0 | |
Toxicity | 4 | |
Body Contact | 3 | |
Reactivity | 0 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Water treatment, biocide.
"water treatment biocide"
Causes burns.
Risk of serious damage to eyes.
May cause SENSITIZATION by skin contact.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.
Accidental ingestion of the material may be seriously damaging to the health of the individual; animal experiments indicate that ingestion of less than 40 gram may be fatal. The material can produce chemical burns within the oral cavity and gastrointestinal tract following ingestion. A metallic taste, nausea, vomiting and burning feeling in the upper stomach region occur after ingestion of copper and its derivatives. The vomitus is usually green/blue and discolors contaminated skin. Acute poisonings from ingestion are rare due to their prompt removal by vomiting. Should vomiting not occur, or is delayed systemic poisoning may occur producing kidney and liver damage, wide-spread capillary damage, and be fatal; death may occur after relapse from an apparent recovery. Anemia may occur in acute poisoning.
If applied to the eyes, this material causes severe eye damage. The material can produce chemical burns to the eye following direct contact. Vapors or mists may be extremely irritating.
The material can produce chemical burns following direct contactwith the skin. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.
Inhalation may produce serious health damage*. Not normally a hazard due to non-volatile nature of product. Inhalation of vapors or aerosols (mists, fumes), generated by the material during the course of normal handling, may produce severe damage to the health of the individual. Relatively small amounts absorbed through the lungs may prove fatal. The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage. Inhalation of quantities of liquid mist may be extremely hazardous, even lethal due to spasm, extreme irritation of larynx and bronchi, chemical pneumonitis and pulmonary edema.
Repeated or prolonged exposure to corrosives may result in the erosion of teeth, inflammatory and ulcerative changes in the mouth and necrosis (rarely) of the jaw. Bronchial irritation, with cough, and frequent attacks of bronchial pneumonia may ensue. Gastrointestinal disturbances may also occur. Chronic exposures may result in dermatitis and/or conjunctivitis. There is limited evidence that, skin contact with this product is more likely to cause a sensitization reaction in some persons compared to the general population. Contact allergies quickly manifest themselves as contact eczema, more rarely as urticaria or Quincke's edema. The pathogenesis of contact eczema involves a cell-mediated (T lymphocytes) immune reaction of the delayed type. Other allergic skin reactions, e.g. contact urticaria, involve antibody-mediated immune reactions. The significance of the contact allergen is not simply determined by its sensitization potential: the distribution of the substance and the opportunities for contact with it are equally important. A weakly sensitizing substance which is widely distributed can be a more important allergen than one with stronger sensitizing potential with which few individuals come into contact. From a clinical point of view, substances are noteworthy if they produce an allergic test reaction in more than 1% of the persons tested.