JARDINE ICON 2.5 EC
Flammability | 2 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 1 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Insecticide.
"pyrethroid insecticide"
Harmful by inhalation.
HARMFUL - May cause lung damage if swallowed.
Irritating to eyes, respiratory system and skin.
Flammable.
Vapors may cause dizziness or suffocation.
Toxic to bees.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.
Accidental ingestion of the material may be damaging to the health of the individual. Considered an unlikely route of entry in commercial/industrial environments. The liquid may produce gastrointestinal discomfort and may be harmful if swallowed. Ingestion may result in nausea, pain and vomiting. Vomit entering the lungs by aspiration may cause potentially lethal chemical pneumonitis. Ingestion of petroleum hydrocarbons can irritate the pharynx, esophagus, stomach and small intestine, and cause swellings and ulcers of the mucous. Symptoms include a burning mouth and throat; larger amounts can cause nausea and vomiting, narcosis, weakness, dizziness, slow and shallow breathing, abdominal swelling, unconsciousness and convulsions. Damage to the heart muscle can produce heart beat irregularities, ventricular fibrillation (fatal) and ECG changes. The central nervous system can be depressed. Light species can cause a sharp tingling of the tongue and cause loss of sensation there. Aspiration can cause cough, gagging, pneumonia with swelling and bleeding.
There is some evidence to suggest that this material can causeeye irritation and damage in some persons. Direct eye contact with petroleum hydrocarbons can be painful, and the corneal epithelium may be temporarily damaged. Aromatic species can cause irritation and excessive tear secretion.
Skin contact with the material may damage the health of the individual; systemic effects may result following absorption. There is some evidence to suggest that this material can cause inflammation of the skin on contact in some persons. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected. Aromatic hydrocarbons may produce sensitivity and redness of the skin. They are not likely to be absorbed into the body through the skin but branched species are more likely to.
If inhaled, this material can irritate the throat andlungs of some persons. If exposure to highly concentrated solvent atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and possible death. Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordination and vertigo. Inhalation hazard is increased at higher temperatures. This material, like natural pyrethrins, may cause central stimulation with nausea, vomiting, stomach upset, diarrhea, hypersensitivity, inco-ordination, tremors, muscle paralysis, convulsion, coma and respiratory failure. Type II compounds cause a "Type II syndrome" characterized by irregular jerky movements, increased saliva production without tears, upper abdominal pain, nausea and vomiting, headache, dizziness, loss of appetite, tiredness, chest tightness, blurred vision, "pins and needles", palpitations, coarse muscle jerks in limbs and altered consciousness. Convulsions can occur in severe cases with flexed arms, extended legs (spastic posture) and unconsciousness. Recovery may take weeks. Acute effects from inhalation of high vapor concentrations may be chest and nasal irritation with coughing, sneezing, headache and even nausea.
There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population.
Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems. There is some evidence that human exposure to the material may result in developmental toxicity. This evidence is based on animal studies where effects have been observed in the absence of marked maternal toxicity, or at around the same dose levels as other toxic effects but which are not secondary non-specific consequences of the other toxic effects. Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys.