K&H 400G ETCH PRIMER (AEROSOL)
Flammability | 4 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 1 | |
Chronic | 3 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Application is by spray atomization from a hand held aerosol pack. Primer surfacer for
adhesion to ferrous and non ferrous metals.
"metal priming paint"
Limited evidence of a carcinogenic effect.
Harmful by inhalation, in contact with skin and if swallowed.
Irritating to eyes and skin.
Extremely flammable.
Vapors may cause dizziness or suffocation.
Risk of explosion if heated under confinement.
Not normally a hazard due to physical form of product. Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. Depression of the central nervous system is the most outstanding effect of most halogenated aliphatic hydrocarbons. Inebriation and excitation, passing into narcosis, is a typical reaction. In severe acute exposures there is always a danger of death from respiratory failure or cardiac arrest due to a tendency to make the heart more susceptible to catecholamines (adrenalin). Considered an unlikely route of entry in commercial/industrial environments. The liquid may produce gastrointestinal discomfort and may be harmful if swallowed. Ingestion may result in nausea, pain and vomiting. Vomit entering the lungs by aspiration may cause potentially lethal chemical pneumonitis. Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.
This material can cause eye irritation and damage in some persons. The material may produce severe irritation to the eye causing pronounced inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.
This material can cause inflammation of the skin oncontact in some persons. Skin contact with the material may be harmful; systemic effects may resultfollowing absorption. Spray mist may produce discomfort. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected. The material may cause severe skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin. Repeated exposures may produce severe ulceration.
Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be harmful. Inhalation of toxic gases may cause: · Central Nervous System effects including depression, headache, confusion, dizziness, stupor, coma and seizures; · respiratory: acute lung swellings, shortness of breath, wheezing, rapid breathing, other symptoms and respiratory arrest; · heart: collapse, irregular heartbeats and cardiac arrest; · gastrointestinal: irritation, ulcers, nausea and vomiting (may be bloody), and abdominal pain.
Principal route of occupational exposure to the gas is by inhalation. Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems. There has been concern that this material can cause cancer or mutations, but there is not enough data to make an assessment. There is some evidence to provide a presumption that human exposure to the material may result in impaired fertility on the basis of: some evidence in animal studies of impaired fertility in the absence of toxic effects, or evidence of impaired fertility occurring at around the same dose levels as other toxic effects but which is not a secondary non- specific consequence of other toxic effects. There is some evidence that human exposure to the material may result in developmental toxicity. This evidence is based on animal studies where effects have been observed in the absence of marked maternal toxicity, or at around the same dose levels as other toxic effects but which are not secondary non-specific consequences of the other toxic effects. Exposure to the material for prolonged periods may cause physical defects in the developing embryo (teratogenesis). Dichloromethane exposures cause liver and kidney damage in animals and this justifies consideration before exposing persons with a history of impaired liver function and/or renal disorders. Dichloromethane is stored in body fat and metabolized to carbon monoxide, which reduces the oxygen carrying capacity of blood.