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K&H FIBREGLASS POLISH WAX MSDS报告[下载][中文版]

Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

K&H FIBREGLASS POLISH WAX

NFPA

Flammability 1
Toxicity 2
Body Contact 2
Reactivity 1
Chronic 2
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4

PRODUCT USE

Polish wax.

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Irritating to eyes and skin.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  The material has NOT been classified as "harmful by ingestion". This is because of the lack of corroborating animal or human evidence. The material may still be damaging to the health of the individual, following ingestion, especially where pre-existing organ (e.g. liver, kidney) damage is evident. Present definitions of harmful or toxic substances are generally based on doses producing mortality (death) rather than those producing morbidity (disease, ill-health). Gastrointestinal tract discomfort may produce nausea and vomiting. In an occupational setting however, unintentional ingestion is not thought to be cause for concern.  

EYE

  This material can cause eye irritation and damage in some persons.  Direct eye contact with petroleum hydrocarbons can be painful, and the corneal epithelium may be temporarily damaged. Aromatic species can cause irritation and excessive tear secretion.  

SKIN

  This material can cause inflammation of the skin oncontact in some persons.  The material may accentuate any pre-existing dermatitis condition.  Irritation and skin reactions are possible with sensitive skin.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  

INHALED

  Inhalation of vapors or aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual.  Acute silicosis occurs under conditions of extremely high silica dust exposure particularly when the particle size of the dust is small. The disease is rapidly progressive and spreads widely through the lungs within months of the initial exposure and causing deaths within 1 to 2 years.  Effects on lungs are significantly enhanced in the presence of respirableparticles.  

CHRONIC HEALTH EFFECTS

  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  This material can cause serious damage if one is exposed to it for long periods. It can be assumed that it contains a substance which can produce severe defects. This has been demonstrated via both short- and long-term experimentation.  There has been some concern that this material can cause cancer or mutations but there is not enough data to make an assessment.  Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys.  Long term exposure to morpholine and some of its congeners may produce liver and kidney damage. Obvious evidence of chronic nasal irritation and inflammation and ocular injury (including retinal degeneration, corneal irritation, uveitis and corneal damage) has been documented in rats exposed to 150 ppm, 6 hours/day, 5 days/week for 104 weeks. Earlier reports linking exposure to morpholine with an increased incidence of hepatocellular carcinoma and pulmonary angiosarcoma, probably resulted from exposure to the carcinogenic contaminant, N-nitrosomorpholine. It must be noted, however, that there is a potential to convert morpholine, a secondary amine), in the body, to the potentially carcinogenic N-  nitros-morpholine. N-nitroso-compounds represent a major class of important chemical carcinogens and mutagens. The induction of tumours by single doses of these substances testify to their potency. Whilst it is difficult to extrapolate animal carcinogenicity data to humans, such data strongly suggests that these compounds are human carcinogens. As a rule the N-nitrosamines as a group are carcinogenic in a multitude of organs and tissues. This is also true for the individual N-nitrosamines where the tumour localisation does not depend only on the kind of nitrosamine but also the species and dose. Mostly, however, a preferred target organ (or even several) can be identified. This is frequently the liver.  
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