Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

JOHNSONDIVERSEY SUMA CLASSIC M7

NFPA

PRODUCT USE

Detergent - dishwashers, dishwashing detergent, dishwashing powder.

SYNONYMS

"HH11059 Suma Classic M7 10kg", "DiverseyLever Divermax M7"

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Harmful if swallowed.
Causes burns.
Risk of serious damage to eyes.
Harmful to aquatic organisms, may cause long- term adverse effects in the
aquatic environment.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  The material can produce chemical burns within the oral cavity and gastrointestinal tract following ingestion.  Ingestion of dichloroisocyanurates will give rise to corrosive attack on the mouth, oesophagus and internal organs and may result in weakness, lethargy, tremors, salivation, lachrymation and possible coma. Toxicity seems to be related to the corrosive effects of the substance (or its aqueous reaction product hypochlorous acid) on the stomach lining, rather than to systemic effects. The severity of these effects seem to be related more to concentration (ie. available chlorine) than to the amount ingested.  Ingestion of alkaline corrosives may produce burns around the mouth, ulcerations and swellings of the mucous membranes, profuse saliva production, with an inability to speak or swallow. Both the esophagus and stomach may experience burning pain; vomiting and diarrhea may follow. Epiglottal swelling may result in respiratory distress and asphyxia; shock can occur. Narrowing of the esophagus, stomach or stomach valve may occur immediately or after a long delay (weeks to years). Severe exposure can perforate the esophagus or stomach leading to infections of the chest or abdominal cavity, with low chest pain, abdominal stiffness and fever. All of the above can cause death.  Single and repeated dose studies in animals by oral and skin routes of cyanuric acid and some cyanurates generally show a low degree of toxicity. At high doses several studies showed kidney damage.  

EYE

  The material can produce chemical burns to the eye following direct contact. Vapors or mists may be extremely irritating.  If applied to the eyes, this material causes severe eye damage.  Direct eye contact with corrosive bases can cause pain and burns. There may be swelling, epithelium destruction, clouding of the cornea and inflammation of the iris. Mild cases often resolve; severe cases can be prolonged with complications such as persistent swelling, scarring, permanent cloudiness, bulging of the eye, cataracts, eyelids glued to the eyeball and blindness.  

SKIN

  The material can produce chemical burns following direct contactwith the skin.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  Skin contact with alkaline corrosives may produce severe pain and burns; brownish stains may develop. The corroded area may be soft, gelatinous and necrotic; tissue destruction may be deep.  

INHALED

  If inhaled, this material can irritate the throat andlungs of some persons.  Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled.  Chlorine vapour is extremely irritating to the upper respiratory tract and lungs  Symptoms of exposure to chlorine include coughing, choking, breathing difficulty, chest pain, headache, vomiting, pulmonary oedema. Inhalation may cause lung congestion, bronchitis and loss of consciousness. Effects may be delayed. Delayed effects of exposure to chlorine vapour can include shortness of breath, violent headaches, pulmonary oedema and pneumonia.  Earlier reports suggested that concentrations around 5 ppm chlorine caused respiratory complaints, corrosion of the teeth, inflammation of the mucous membranes of the nose and increased susceptibility to tuberculosis in chronically-exposed workers. Recent studies have not confirmed these findings. Concentrations too low to effect the lower respiratory tract may however irritate the eyes, nose and throat.  Amongst 29 volunteers exposed at 0.5, 1 or 2 ppm chlorine for 4 to 8 hours the following responses were recorded: itching or burning of the nose, itching or burning of the throat,  production of tears, urge to cough, runny nose, nausea, headache, general discomfort, dizziness, drowsiness and shortness of breath.  Inhaling corrosive bases may irritate the respiratory tract. Symptoms include cough, choking, pain and damage to the mucous membrane. In severe cases, lung swelling may develop, sometimes after a delay of hours to days. There may be low blood pressure, a weak and rapid pulse, and crackling sounds.  

CHRONIC HEALTH EFFECTS

  Repeated or prolonged exposure to corrosives may result in the erosion of teeth, inflammatory and ulcerative changes in the mouth and necrosis (rarely) of the jaw. Bronchial irritation, with cough, and frequent attacks of bronchial pneumonia may ensue. Gastrointestinal disturbances may also occur. Chronic exposures may result in dermatitis and/or conjunctivitis.  Long-term exposure to respiratory irritants may result in disease of the airways involving difficult breathing and related systemic problems.  Reduced respiratory capacity may result from chronic low level exposure to chlorine gas. Chronic poisoning may result in coughing, severe chest pains, sore throat and haemoptysis (bloody sputum). Moderate to severe exposures over 3 years produced decreased lung capacity in a number of workers.  Delayed effects can include shortness of breath, violent headaches, pulmonary oedema and pneumonia.  Amongst chloralkali workers exposed to mean concentrations of 0.15 ppm for an average of 10.9 years a generalised pattern of fatigue (exposures of 0.5 ppm and above) and a modest increased incidence of anxiety and dizziness were recorded. Leukocytosis and a lower haematocrit showed some relation to exposure.  The chlorinated isocyanurates have low acute oral and dermal toxicity but are very irritating to the eyes, They are very mild skin irritants and are not considered to be skin sensitizers.  A subchronic toxicity study showed effects in the urinary bladders of male mice and rats. Chronic toxicity studies (2-year feeding studies) using rats and mice showed no oncogenic effects at any dose level. The chlorinated isocyanurates are not teratogenic or mutagenic. Metabolism studies show that they are rapidly absorbed, distributed and excreted unmetabolised.  Chronic inhalation toxicity: Rats were exposed to sodium dichloroisocyanurate dihydrate dust at 3, 10 and 30 mg/m3 for 6hrs/day, 5/days/week for 4 weeks and the 10 and 30 mg/m3 groups showed some signs of toxicity (eye and respiratory tract irritation, reduced growth and altered organ weights) [Monsanto, Kirk-Othmer]  Chronic ingestion toxicity: Signs of severe toxicity (increased deaths, difficult breathing, gastrointestinal bleeding and reduced activity and growth) were seen in rats fed on high doses of dichloroisocyanurate in drinking water (4000 and 8000 ppm) for 59 days. Rats fed high doses of dichloroisocyanurate (6000 and 12000 ppm) in th diet for 13 weeks showed some signs of toxicity (reduced growth and reduced liver end kidney weights. [Monsanto, Kirk-Othmer]  Delayed effects can include shortness of breath, violent headaches, pulmonary oedema and pneumonia. Experimental studies on laboratory animals indicate possible teratogenic and other reproductive effects. [BASF].  Long term exposure to high dust concentrations may cause changes in lung function i.e. pneumoconiosis; caused by particles less than 0.5 micron penetrating and remaining in the lung. Prime symptom is breathlessness; lung shadows show on X-ray.  Exposure to the material for prolonged periods may cause physical defects in the developing embryo (teratogenesis).