HYDROXYETHYL CETYLDIMONIUM PHOSPHATE
Flammability | 1 | |
Toxicity | 2 | |
Body Contact | 3 | |
Reactivity | 1 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Cationic surfactant.
C20H44NOH2O4P, C20-H46-N-O5-P, C20-H46-N-O5-P, "cetyl dimethyl-2-hydroxyethyl ammonium
dihydrogen phosphate", "cetyl dimethyl-2-hydroxyethyl ammonium dihydrogen phosphate", "1-
hexadecammonium, N-(2-hydroxyethyl)-N, N-dimethyl-, phosphate (1:1) salt", "1-
hexadecammonium, N-(2-hydroxyethyl)-N, N-dimethyl-, phosphate (1:1) salt",
"cetyldimethyl(2-hydroxyethyl)ammonium dihydrogen phosphate", "Luviquat Mono CP",
"quaternary ammonium"
Harmful if swallowed.
Irritating to eyes and skin.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.
Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. Concentrated solutions of many cationics may cause corrosive damage to mucous membranes and the esophagus. Nausea and vomiting (sometimes bloody) may follow ingestion. Serious exposures may produce an immediate burning sensation of the mouth, throat and abdomen with profuse salivation, ulceration of mucous membranes, signs of circulatory shock (hypotension, labored breathing, and cyanosis) and a feeling of apprehension, restlessness, confusion and weakness. Weak convulsive movements may precede central nervous system depression. Erosion, ulceration, and petechial hemorrhage may occur through the small intestine with glottic, brain and pulmonary edema. Death may result from asphyxiation due to paralysis of the muscles of respiration or cardiovascular collapse. Fatal poisoning may arise even when the only pathological signs are visceral congestion, swallowing, mild pulmonary edema or varying signs of gastrointestinal irritation. Individuals who survive a period of severe hypertension may develop kidney failure. Cloudy swelling, patchy necrosis and fatty infiltration in such visceral organs as the heart, liver and kidneys shows at death.
This material can cause eye irritation and damage in some persons. If applied to the eyes, this material causes severe eye damage.
The material may cause moderate inflammation of the skin either following direct contact or after a delay of some time. Repeated exposure can cause contact dermatitis which is characterized by redness, swelling and blistering. Skin contact with the material may damage the health of the individual; systemic effects may result following absorption. Solution of material in moisture on the skin, or perspiration, may markedly increase skin corrosion and accelerate tissue destruction. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.
The material is not thought to produce either adverse health effects or irritation of the respiratory tract following inhalation (as classified using animal models). Nevertheless, adverse effects have been produced following exposure of animals by at least one other route and good hygiene practice requires that exposure be kept to a minimum and that suitable control measures be used in an occupational setting.
There is limited evidence that, skin contact with this product is more likely to cause a sensitization reaction in some persons compared to the general population. Long term exposure to high dust concentrations may cause changes in lung function i.e. pneumoconiosis; caused by particles less than 0.5 micron penetrating and remaining in the lung. Prime symptom is breathlessness; lung shadows show on X-ray.