WHITE KNIGHT NATURAL OIL TIMBER DRESSING
Flammability | 2 | |
Toxicity | 2 | |
Body Contact | 3 | |
Reactivity | 1 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Finish for brush application on timber surfaces. The use of a quantity of material in an
unventilated or confined space may result in increased exposure and an irritating
atmosphere developing.Before starting consider control of exposure by mechanical
ventilation.
"alkyd resin solution", "clear tints tinted colours fungicidal top coat"
Risk of serious damage to eyes.
May cause SENSITIZATION by skin contact.
Limited evidence of a carcinogenic effect.
Possible risk of harm to the unborn child.
HARMFUL - May cause lung damage if swallowed.
Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Harmful by inhalation and if swallowed.
Irritating to respiratory system and skin.
Flammable.
Vapors may cause dizziness or suffocation.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.
Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. Considered an unlikely route of entry in commercial/industrial environments. Ingestion may result in nausea, pain, vomiting. Vomit entering the lungs by aspiration may cause potentially lethal chemical pneumonitis.
If applied to the eyes, this material causes severe eye damage. The liquid may produce eye discomfort and is capable of causing temporary impairment of vision and/or transient eye inflammation, ulceration. The material may produce severe irritation to the eye causing pronounced inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.
This material can cause inflammation of the skin oncontact in some persons. Skin contact is not thought to have harmful health effects, however the material may still produce health damage following entry through wounds, lesions or abrasions. The liquid may produce skin discomfort following prolonged contact. Defatting and/or drying of the skin may lead to dermatitis. The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin.
If inhaled, this material can irritate the throat andlungs of some persons. Inhalation hazard is increased at higher temperatures. Acute effects from inhalation of high vapor concentrations may be chest and nasal irritation with coughing, sneezing, headache and even nausea. If exposure to highly concentrated solvent atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and possible death. Inhalation of vapor may aggravate a pre-existing respiratory condition such as asthma, bronchitis, emphysema.
There has been concern that this material can cause cancer or mutations, but there is not enough data to make an assessment. Skin contact with the material is more likely to cause a sensitization reaction in some persons compared to the general population.
Principal routes of exposure are usually by inhalation of vapor and skin contact. Prolonged or continuous skin contact with the liquid may cause defatting with drying, cracking, irritation and dermatitis following. Chronic solvent inhalation exposures may result in nervous system impairment and liver and blood changes. [PATTYS]. Ingestion may produce nausea, vomiting and diarrhea, bloody stools, shock, rapid pulse and coma. Severe gastritis or gastroenteritis may occur as a result of lesions produced by vascular damage from absorbed arsenic (and not local corrosion); symptoms may be delayed for several hours. Eventually a violent hemorrhagic gastroenteritis leads to profound loss of fluid and electrolyte resulting in shock and death. Occasionally alimentary symptoms are mild or absent in which case symptoms are usually referable to the central nervous system, headache, vertigo, muscle spasm or convulsion, delirium and, sometimes, mania. In advanced poisonings by arsenic and its inorganic salts, nervous symptoms are prominent; disorders of the brain (encephalopathies) and peripheral neuritis (more commonly) have been described. A prickling sensation (paresthesia), decreased sensitivity to sensation and pain (hypoesthesia), eventually paralysis and muscular atrophy appear, usually in the legs. "Glove and stocking' distribution of sensory loss may be prominent. The toxic moiety is presumed to be trivalent arsenic in the form of inorganic arsenious acid (arsenite) or an organic arsenoxide. Arsenites are active enzyme inhibitors. Arsenic and its compounds may damage the stem cell which acts as the precursor to components of the blood. Loss of the stem cell may result in pancytopenia (a reduction in the number of red and white blood cells and platelets) with a latency period corresponding to the lifetime of the individual blood cells. Granulocytopenia (a reduction in granular leukocytes) develops within days and thrombocytopenia (a disorder involving platelets), within 1-2 weeks, whilst loss of erythrocytes (red blood cells) need months to become clinically manifest. Aplastic anaemia develops due to complete destruction of the stem cells.