Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

WATTYL HANDI LOW SHEEN ACRYLIC

NFPA

PRODUCT USE

Used according to manufacturer' s directions.

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Toxic to aquatic organisms.
Harmful to aquatic organisms, may cause long- term adverse effects in the
aquatic environment.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Toxic effects may result from the accidental ingestion of the material; animal experiments indicate that ingestion of less than 40 gram may be fatal or may produce serious damage to the health of the individual.  In humans, inorganic nitrites produce smooth muscle relaxation, methaemoglobinaemia (MHG) and cyanosis. Fatal poisonings in infants, resulting from ingestion of nitrates in water or spinach, have been reported. The primary effect of nitrite intoxication in animals is MHG whilst secondary effects include vasodilation, relaxation of smooth muscle and lowering of blood pressure. Other nitrite-induced toxic effects include abdominal pain, diarrhoea, atrophied intestinal villi and apoptopic cell death in the intestinal crypts. When sodium nitrite was administered in drinking water for 6 weeks (0.06-1%), mice showed a slight degeneration and spotty necrosis of hepatocytes and haemosiderin deposition in the liver, spleen and lymph nodes, indicating haemolysis. At 2%, mice died within 3 weeks. In rats, subject to the same treatment regime, abnormal blood and spleen colours, due to MHG, were seen in 0.5% and 1.0% treatment groups. Hepatic microsomal lipoperoxidation (as measured by malondialdehyde formation) was increased in male rats given 0.2% sodium nitrite in drinking water. Liver lysosomal enzymes (acid phosphatase aaand cathepsin) and superoxide dismutase activities were also increased. This data suggests that the nitrite stimulates generation of superoxide radicals in the liver causing damage to cellular and subcellular membranes. Decreased plasma vitamin E and greater reduced glutathione-per erythrocyte were also reported in male rats receiving sodium nitrite in drinking water.  

EYE

  The material may produce moderate eye irritation leading to inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.  There is some evidence to suggest that this material can causeeye irritation and damage in some persons.  

SKIN

  There is some evidence to suggest that this material can cause inflammation of the skin on contact in some persons.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  

INHALED

  The material is not thought to produce either adverse health effects or irritation of the respiratory tract following inhalation (as classified using animal models). Nevertheless, adverse effects have been produced following exposure of animals by at least one other route and good hygiene practice requires that exposure be kept to a minimum and that suitable control measures be used in an occupational setting.  

CHRONIC HEALTH EFFECTS

  There is limited evidence that, skin contact with this product is more likely to cause a sensitization reaction in some persons compared to the general population.  Sensitization may result in allergic dermatitis responses includingrash, itching, hives or swelling of extremities.  Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems.  There has been some concern that this material can cause cancer or mutations but there is not enough data to make an assessment.