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UOP HC-T-1.3Q UNIONFINING CATALYST MSDS报告[下载][中文版]

Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

UOP HC-T-1.3Q UNIONFINING CATALYST

NFPA

Flammability 0
Toxicity 2
Body Contact 2
Reactivity 0
Chronic 4
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4

PRODUCT USE

Catalyst.

SYNONYMS

catalyst

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Danger of cumulative effects.
May cause CANCER by inhalation.
May cause SENSITIZATION by skin contact.
Harmful: danger of serious damage to health by prolonged exposure through
inhalation and if swallowed.
Irritating to eyes and respiratory system.
May cause long- term adverse effects in the aquatic environment.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Considered an unlikely route of entry in commercial/industrial environments.  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  Nickel salts cause vomiting, following ingestion as a result of the irritant effects. Absorption is generally poor and systemic poisoning is rare. Systemic effects include increased blood sugar levels, capillary damage, kidney damage, heart damage and central nervous system depression.  

EYE

  This material can cause eye irritation and damage in some persons.  

SKIN

  The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  Sensitization may result in allergic dermatitis responses includingrash, itching, hives or swelling of extremities.  

INHALED

  The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.  

CHRONIC HEALTH EFFECTS

  Repeated or long-term occupational exposure is likely to produce cumulative health effects involving organs or biochemical systems.  Asthma-like symptoms may continue for months or even years after exposure to the material ceases. This may be due to a non-allergenic condition known as reactive airways dysfunction syndrome (RADS) which can occur following exposure to high levels of highly irritating compound. Key criteria for the diagnosis of RADS include the absence of preceding respiratory disease, in a non-atopic individual, with abrupt onset of persistent asthma-like symptoms within minutes to hours of a documented exposure to the irritant. A reversible airflow pattern, on spirometry, with the presence of moderate to severe bronchial hyperreactivity on methacholine challenge testing and the lack of minimal lymphocytic inflammation, without eosinophilia, have also been included in the criteria for diagnosis of RADS. RADS (or asthma) following an irritating inhalation is an infrequent disorder with rates related to the concentration of and duration of exposure to the irritating substance. Industrial bronchitis, on the other hand, is a disorder that occurs as result of exposure due to high concentrations of irritating substance (often particulate in nature) and is completely reversible after exposure ceases. The disorder is characterised by dyspnea, cough and mucus production.  There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population.  Skin contact with the material is more likely to cause a sensitization reaction in some persons compared to the general population.  There is sufficient evidence to suggest that this materialdirectly causes cancer in humans.  Exposure to large doses of Aluminum has been connected with the degenerative brain disease Alzheimer's Disease.  High levels of molybdenum can cause joint problems in the hands and feet with pain and lameness. Molybdenum compounds can also cause liver changes with elevated levels of enzymes and cause over-activity of the thyroid gland. A generalized feeling of unwellness can occur, with tiredness, weakness, diarrhea, loss of appetite and weight. Molybdenum has been associated with cancers of the airways, but on the other hand, a low intake of molybdenum may cause an increased risk of developing esophageal cancer.  Repeated exposures, in an occupational setting, to high levels of fine- divided dusts may produce a condition known as pneumoconiosis which is the lodgement of any inhaled dusts in the lung irrespective of the effect. This is particularly true when a significant number of particles less than 0.5 microns (1/50,000 inch), are present. Lung shadows are seen in the X-ray. Symptoms of pneumoconiosis may include a progressive dry cough, shortness of breath on exertion, increased chest expansion, weakness and weight loss. As the disease progresses the cough produces a stringy mucous, vital capacity decreases further and shortness of breath becomes more severe. Pneumoconiosis is the accumulation of dusts in the lungs and the tissue reaction in its presence. It is further classified as being of noncollagenous or collagenous types. Noncollagenous pneumoconiosis, the benign form, is identified by minimal stromal reaction, consists mainly of reticulin fibres, an intact alveolar architecture and is potentially reversible.  Nickel causes a skin sensitization which may produce a chronic eczema. At first an itch appears followed one week later by a red skin eruption with ulcers which discharge and become crusted. In the chronic stages, pigmented or depigmented plaques may be formed. Recovery from the skin inflammation may take weeks. Nickel dusts and some of its compounds may cause cancer; nickel workers show an increased risk of developing cancers of the lung and nasal cavity.  
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